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Wednesday, May 3, 2017

History of the Zika Virus

(By [http://EzineArticles.com/expert/Norma_Holt/270038]Norma Holt)

The name comes from the Zika forest in Uganda where in 1947 researchers into Yellow Fever infected a rhesus macaque with the fever. They isolated a transmissible substance in 1952, called the Zika virus, and took substance from the first infected human of Nigeria in 1954. From then until 2007 cases of it were rare in Africa and Asia until a major outbreak occurred on Yap island, Micronesia. Since then it has been found in Polynesia, Easter Island, the Cook Islands and New Caledonia.

The question is who created the virus and is responsible for its spread? Obviously the monkey must have passed on the infection through mosquitoes in its area and to others in its pack. Then it passed to humans while the scientists may have allowed the substance they created to pass into the environment.

It doesn't matter how it came to be or why the facts are we have it and now it is set to play havoc on human health. In the intervening 62 years it has travelled from Africa and Asia to Brazil and now to neighbouring countries like Venezuela, Argentina, and so on. In 2015 this larger outbreak in Brazil is linked to the staging of the FIFA World Cup in 2014.

The facts are that humans are subject to something beyond their control. It is my opinion that we are in the last days when life on earth will almost disappear and only a few will survive. This is not based on speculation but on memory of my reincarnation and knowledge given to me by the Spirit of the Universe, the real God.

It is this entity that controls all things and it is in all of space. Look up and see the zillions upon zillions of stars and planets to get an idea of its size. It can also speak to us and put in our minds the things it requires to happen, such as creating this virus. With its spread devastation on a massive scale will follow as it causes microcephaly in infants and that may lead to another greater disaster.

The virus is also known to stay in the semen and to be transmitted to women, as noted by officials at the Dallas County Health and Human Services and confirmed by the Centers for Disease Control and Prevention on February 2nd.

If this is the scenario we are faced with there is little we can do about it. The Olympic Games will go ahead as planned and the virus will spread because, as noted in prophecy, we are caught in a trap (Jeremiah 11:11,12) from which there is no escape.

Norma Holt has memory of her reincarnation she knows that [http://reincarnationfacts.com]reincarnation is fact and that everyone who has lived is back. It is the time of judgment and they are turning to the [http://mountaintalk.net]Mountain of God which is the Internet, promised for this time, for answers.

Article Source: [http://EzineArticles.com/?History-of-the-Zika-Virus&id=9435814] History of the Zika Virus

Autoimmune Disease: Your Body At War With Itself

(By [http://EzineArticles.com/expert/Terry_Lynne_Hale/19609]Terry Lynne Hale)

I've been searching for alternatives (alternatives to prescribed drugs, to foods, to attitude) for a very long time. A couple of decades are a long time to feel generally awful; without vibrancy, without satisfactory energy, without a clear head. People who don't know me very well or haven't known me very long are surprised to hear this. To them, I appear high energy; I move quickly, speak fast, and can be very reactive. I accomplish a great deal.

I know I could accomplish so much more if given the energy, stamina, and vitality to combine with the enthusiasm for life that I have.

In my search for more, I learned some incredible facts about autoimmunity. According to the National Institute of Allergy and Infectious Diseases, (NIAID) there are more than 80 diseases or disorders attributed to an autoimmune condition. The NIAID maintains the study of autoimmunity is a priority for their organization.

At the turn of the 20th Century - it is estimated that about 1 in 10,000 individuals had an autoimmune disorder. Today, that number is 1 in 250! That's an extraordinary increase in 116 years. In addition to Hypothyroidism (low thyroid hormone) and Hyperthyroidism (too much thyroid hormone) here are some other familiar names of diseases I didn't realize were considered "autoimmune:"

• Parkinson's Disease
• Dementia
• Colitis
• Arthritis
• Irritable bowel syndrome
• Addison's Disease
• Autoimmune hepatitis
• Celiac Disease
• Chronic Lyme's disease
• Restless Legs
• Rheumatoid Arthritis

The list goes on and on.

From what I've learned, inflammation is at the heart of autoimmunity. Inflammation is said to begin in the gut. The gut has a semi-permeable lining whose degree of permeability fluctuates in response to an enormous number of conditions. The result of repeated stretching of this lining is what's known as "leaky gut syndrome."

Leaky gut means partially undigested foods, viruses, bacteria and toxins flow freely into our bloodstream, ultimately leading to inflammation and a severe lack of nutrient absorption. During one of my numerous searches for ways to feel better, I began taking a large number and variety of vitamins. After a while (and a whole lot of money) I learned I probably had a leaky gut. Translation: all the vitamins I was consuming resulted in "expensive urine" and little else.

As of this writing, I'm awaiting lab results. Last week I asked my doctor to order the following 6 labs (vs. the standard 3 typically ordered) so I could find out more about my autoimmune condition:

• TSH (thyroid stimulating hormone)
• Thyroid Peroxidase Thyroid Antibodies (TPO)
• Thyroglobulin Antibodies (TG Antibodies)
• Free T3
• Free T4
• Reverse T3

Some experts also suggest a thyroid ultrasound and I might also request this if I don't obtain sufficient information and direction. There is a ton of information and a huge number of authorities on autoimmune diseases and disorders. The main takeaway I can share from all of my studies is this:

Each of us is responsible for our own health. It is your responsibility to ask the doctors to explain, ask the pharmacist to help you understand your medications, and most of all, gauge your progress. I should have acted long before now to be proactive about my own health. I encourage everyone to do the same.

If you'd like to read more on this subject or on other areas of interest to the author (the Internet, interviewing, business, health, wellness, pets, environment, plus,) visit her site at http://www.terrylynnehale.com or her blog at http://terrylynnehale.com/terry-lynnes-take/

Article Source: [http://EzineArticles.com/?Autoimmune-Disease:-Your-Body-At-War-With-Itself&id=9583974] Autoimmune Disease: Your Body At War With Itself

I Look Like I'm 23, But I'm Really 52

(By [http://EzineArticles.com/expert/Leah_Barker/2347652]Leah Barker)

Often times when I talk about advancing into the later stages of Huntington's Disease, people (for a reason I will never know) think that it is comforting to tell me that I might die by getting ran over a bus, or inheriting breast cancer, or tripping on a rock and cracking my head open. They tell me this to say, "Hey, you never know what the future might bring. You might die from something else before you even progress in the disease!"

Okay... what? Almost every person that I confide in tells me things like this. Let me be the first to say that it is not comforting at all. The fact is, I know that I have HD; I know that my brain is degenerating and that someday, it will destroy my mind and my body.

I may look like I'm 23, but in reality, I'm 52.

On a timeline, that is. My clock is ticking faster than yours, whether you want to believe it or not. I don't have a potential long-life span. This is it. It's inevitable. And I'm okay (for the most part).

What most people don't understand or refuse to believe is that I am sick now. With a CAG of 43, I didn't think that my symptoms would start showing until my 40's, but after doing some research, I found that most people with a CAG of 43 begin to endure emotional problems within their 20's.

This is where my depression, anxiety, paranoia, and mild OCD come in. It's so hard to deal with it all, especially when people believe that it's "all in my head." I am speaking for all the other HD victims who are waging the same war. And it is, indeed, a blood-shedding battle that we fight every day, while everyone else goes about their routine business.

On the days that I cannot get out of bed and I have to keep the curtains shut, I am not being lazy. I am fighting the demons in my head that tell me that dying would be better than progressing, and let me tell you, I cannot imagine this mental degradation getting worse, but it has to.

When I don't answer my phone calls or talk to my friends when they want to hang out, it is not because I'm being anti-social. It is because I have days when I'm afraid of crowds or going outside. I'm afraid of the sunlight because it reminds me of a happiness that is slipping further from my grip every day. I'm almost always afraid of something, but it's just that word: something. I don't know what it is or when it will hit. It comes and goes as it pleases, and it steals every ounce of will and inspiration that I cling so desperately to on the good days.

I know there are others who are going through the same thing, and I'm so thankful for the fellow victims who take my hand and suffer with me.

I just want to announce that this is a disease, not a choice. I can only control so much, and it kills me.

My brain is like a big jigsaw puzzle that loses more pieces as the clock keeps ticking. Sometimes, on rare occasions, I think that the puzzle might be complete, but the euphoria only lasts for so long, and then the pieces become lost again.

But I'm going to embrace it. The manic depression, anxiety, paranoia, bipolar disorder, and slight schizophrenia-they are all a part of me, and though they may appear as enemies, I like to call them my friends when they allow me to relate to others who are wading through similar waters.

So, I am going to call them my friends for as long as I have the will, and I plan on having the will for a long, long time. Maybe until my very last breath. Only time will tell.

Leah Barker
Capturingthecorners.com
We Want to Hear Your Story. Visit http://capturingthecorners.com/tell-your-story/

Article Source: [http://EzineArticles.com/?I-Look-Like-Im-23,-But-Im-Really-52&id=9562424] I Look Like I'm 23, But I'm Really 52

Cord Blood Stem Cells Show Promise for Alzheimer's Patients



(Cord Blood Stem Cells Show Promise for Alzheimer's Patients
By [http://EzineArticles.com/expert/Richa_Verma/2314151]Richa Verma)

Alzheimer's disease affects almost 4 million Indians and is predicted to triple its numbers by 2050. A disease that impacts basic cerebral functions like memory and thinking skills, it is irreparable and currently has no cure or treatment available. Latest scientific developments involve the encouraging promise of the restorative usage of cord blood stem cells in curing this progressive brain disease.

Neuroscientists from the University of South Florida, USA led by Dr. Jun Tan, MD, PhD, Robert A. Silver Chair and Director of the Rashid Laboratory for Developmental Neurobiology at the Silver Child Development Centre, USF Department of Psychiatry have been bestowed a federal grant of USD 1.5 million in order to assess a new methodology of treatment in a mouse model for Alzheimer's disease using human umbilical cord blood cells (HUCBC) for a period of 5 years by National Institute on Aging. In a mouse model, investigative studies by this team of researchers have demonstrated that immunity can be transmitted using human umbilical cord blood cells, which betters the pathology connected with Alzheimer's disease. Alternatively, additional research work has shown that a particular immune system repression is associated with notably diminished anomalous concentrations of the beta amyloid protein related to Alzheimer's disease.

"This new NIH study will continue to build on our understanding of the HUCBC's mechanism behind improvement in Alzheimer's disease," Dr. Tan said. "It will help provide a better understanding of brain immune cells called microglia, which promote brain inflammation in Alzheimer's disease."

Dr. Tan and his research team comprising of Paul Sanberg, PhD, DSc, Director of the Centre of Excellence for Aging and Brain Repair, Department of Neurosurgery and Brain Repair, and David Morgan, PhD, professor in the Department of Molecular Pharmacology and Physiology and chief scientific officer of the Byrd Alzheimer Institute at USF Health revealed that certain molecule, CD40 on the exterior of these microglia cells is triggered by its partner, CD40L (CD40 ligand) that prompts a series of cascading events that eventually leads to brain inflammation potentially causing injury to the brain's neurons. However, these eventualities resulting in damaging the immune response system could be obstructed by particular antibodies.

In order to reduce the Alzheimer's pathology in the brain, Dr. Tan and his team proposed to examine the prognosis that human umbilical cord blood cells has the potential to diminish the interaction between the CD40L molecule and its CD40 target. A mouse model trial will be executed with genetically altered memory problems imitating Alzheimer's disease as they age.

Moreover, the research team believes that by generating a cocktail amalgamation of the specific molecules would be the fundamental reasons behind the favourable outcomes of the human umbilical cord blood cells in demonstrating its possibility in treating Alzheimer's.

"We will give the compound to these transgenic mice to assess the possibility of bypassing the need for HUCBC and making future therapies more cost effective," Dr. Tan said. "This approach shifts the focus from treating symptoms of Alzheimer's disease to treatments that slow down the disease or prevent it altogether." Dr. Tan said. "Our long-term goal is to move this combination treatment into phase I human trials for patients with mild to moderate this disease."

For details visit   [https://www.cordlifeindia.com/blog/alzheimers-and-stem-cell-therapy/]https://www.cordlifeindia.com/blog/alzheimers-and-stem-cell-therapy/.

Article Source: [http://EzineArticles.com/?Cord-Blood-Stem-Cells-Show-Promise-for-Alzheimers-Patients&id=9536116] Cord Blood Stem Cells Show Promise for Alzheimer's Patients

Bone Diseases and Treatment Process



(Bone Diseases and Treatment Process
By [http://EzineArticles.com/expert/Prachi_Tyagi/2016770]Prachi Tyagi)

Bone disease known as "osteopathy" the term osteopathy is commonly used to refer to another healthcare philosophy. the bones support the body move, shape and help your body. the Living tissues that rebuild constantly throughout your life. During childhood to your teens, the body adds new bone faster than it removes aged bone. After on age 20, you can drop bone faster than you build bone. To have muscular bones when you are young, and to stop bone loss when you are older.

Need to get sufficient vitamin D, calcium and exercise. You should also keep away from drinking too much alcohol and smoking.

Let's start here telling about types of bone diseases:


Osteoporosis- A medical situation in which the bones develop into fragile and Delicate from loss of tissue, typically as a result of hormonal changes, or deficiency of calcium or vitamin D

Osteogenesis imperfecta - An inherited disorder characterized by rearward fragility of the bones

Rickets - A disease of children caused by vitamin D deficiency, characterized by imperfect calcification, softening, and distortion of the bones typically resulting in bow legs.

Fracture of bone- Fracture, in pathology, a break in a bone caused by stress. Certain normal and pathological conditions may predispose the bone to fracture. Children have relatively weak bones due to incomplete calcification, and older adults, especially women past menopause develop osteoporosis, a weakening of concomitant bone with aging.

Osteomyelitis- Infection of bone tissue. The disease is most commonly caused by Staphylococcus aureus infectious organism, reaching the bone through the bloodstream or extension of a local lesion; Inflammation continues with cancellous ossicle destruction (porous) and bone, loss of blood supply and bone death

Osteosarcoma- The most common bone cancer, which affects mainly the long ossicle, particularly the regions knee, hip or shoulder. The cause of osteosarcoma is unknown, but genetic factors and radiotherapy may be involved in its development. Osteosarcoma occurs more often in males than in females; the most affected people are under 30 years.

Bone disease- Any of the diseases or injuries affecting human bones. Diseases and injuries of the bones are the main causes of abnormalities of the human skeletal system. Although physical injury, causing fracture, dominates disease, fracture is just one of the common causes of bone disease, and the disease is in fact a common cause of fracture.

Metabolic bone disease - Any of various diseases causing various abnormalities or ossicle deformities. Examples of metabolic bone diseases including osteoporosis, rickets, osteomalacia, osteogenesis imperfecta, marble bone disease (osteopetrosis), Paget's disease of bone and fibrous dysplasia. In clinical terms, metabolic bone diseases can cause bone pain and loss of height (due to compression of the vertebrae), and predispose patients to fracture.

Achondroplasia - genetic disorder characterized by an abnormality in the conversion of cartilage into ossicle. Consequently, the bones dependent models of cartilage development, particularly the long bones such as the femur and humerus, cannot grow. Achondroplasia is the most common cause of dwarfism.

Neurofibromatosis- Any two distinct hereditary disorders characterized by skin lesions and benign tumors, which are enlarged progressively nervous system. Neurofibromatosis type 1, also known as von Recklinghausen's disease is much more common of the two disorders and is present in approximately one of every 3,000 live births.

Paget disease of bone- chronic disease of middle age, characterized by excessive breakdown and formation of bone tissue. It is a localized disease that can be unifocal, affecting a single bone, or multifocal, affecting many bones or nearly entire skeleton. For this reason, it is included between metabolic ossicle diseases.

Osteomalacia- Condition in which the ossicle of an adult gradually soften due to inadequate bone mineralization. (In children, the condition is called rickets.) Osteomalacia may occur after several pregnancies or old age, resulting in increased susceptibility to fractures. Symptoms include ossicle pain, weakness, numbness of the limbs.

Bone cancer- Disease characterized by the uncontrolled growth of ossicle cells. The primary bone cancer, that is, cancer that arises in the bone directly, is relatively rare. In the United States, for example, only about 2,400 new cases of primary bone cancer are diagnosed each year.

Ewing tumor of bone- This type of bone cancer most commonly appears on the shafts of long bones such as the femur, tibia or humerus, or ribs or flat ossicle. The pelvis, scapula or skull. Related tumors may also develop in soft tissues.

Marble bone disease- Rare disorder in which the bones become extremely dense, hard and brittle. The disease progresses as bone growth continues; Ossiclee cavities are filled with compact bone. Because increased crowds marrow bone mass, resulting in a reduced amount of bone and therefore a reduced capacity to produce red blood cells, severe anemia results capacity.

Osteochondroma - Solitary benign tumor that consists of cartilage and ossicle portion. Osteochondroma are common and can spontaneously develop after a trauma or may have a hereditary basis. No treatment is required unless the tumor interferes with the function, in which case it must be surgically removed.

Osteochondrosis- Temporary orthopedic relatively common disorder in which children epiphysis (end growth) of a bone dies and then gradually replaced over a period of years. The immediate cause of bone death is the loss of blood supply, but the cause of the latter is unclear. The most common form, the flat coxa, or Legg-Calv�-Perthes disease affects the hip.

Fibrous dysplasia- Rare congenital disorder of development that begins in childhood and is characterized by the substitution of solid calcified bone with fibrous tissue, often only on one side of the body and especially in the long ossicle and the pelvis. The disease appears to result from a genetic mutation that leads to overproduction of fibrous tissue.

Cleidocranial dysostosis- congenital disorder, characterized by rare inherited clavicles that are absent or reduced in size, cranial abnormalities and abnormal dentition. The shoulders can sometimes play in front of the chest, and certain facial bones are underdeveloped or missing.

Osteoma- Small, often solitary bone tumor mainly found in the ossicle of the skull. Osteomas generally appear in late childhood or young adulthood; They are often asymptomatic. not become malignant, and treatment (excision) is necessary only if the tumor interferes with normal operation.

Osteoclastoma- Bone is predominantly at the end of long ossicle in the knee region, but also occur in the wrist, arm and pelvis. Large multinucleated cells (giant cells) found in these tumors resemble osteoclasts, so the name of the tumor. Usually seen in adult women between the ages of 20 and 40, this relatively rare, painful tumor is potentially malignant.

Bone cyst - Benigno saclike and that is usually filled with fluid. The unicameral ossicle cyst affect the long bones, particularly the humerus and femur, or heel bones in children and adolescents and is often detected as a result of a fracture. Treatment involves removal of the cyst and a bone graft, but spontaneous healing is common.

Melorheostosis- rare disorder of unknown cause in which cortical bone growth occurs along the main axis of a bone so that resemble drops of candles. Pain is the main symptom, and stiffness and deformity may result. Usually, only one limb and hip or shoulder are affected.

Callus osteology- In osteology, cartilaginous bone material and forming a bridge connecting through a ossicle fracture during repair. Within one to two weeks after injury, a provisional callus forms, wrapping the fracture site. Osteoblasts, bone-forming cells in the periosteum (the layer where new bone bone occurs) proliferate rapidly.

Caffey syndrome- An inherited disease of children, characterized by swelling of the periosteum (bone layer where new ossicle is produced) and the ossicle cortex of the arms, shoulder girdle and lower jaw. The disease is accompanied by fever and irritability; After a series of periodic exacerbations, it disappears spontaneously.

Mandibulofacial dysostosis- A rare genetic disorder, inherited as an autosomal dominant trait and characterized by some or all of the following: underdeveloped cheek ossicle and jaw, widely spaced eyes, malformation of the lower eyelid and lack of eyelashes, malformation of the atrium ear, absence of an external ear canal resulting conductive deafness.

Fracture dislocation- A serious injury fracture and dislocation simultaneously occur. Often, a loose piece of bone remains jammed between the ends of bones and dislocated may have to be surgically removed before displacement can be reduced.


Treatment Process -

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Article Source: [http://EzineArticles.com/?Bone-Diseases-and-Treatment-Process&id=9565738] Bone Diseases and Treatment Process